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Overexpression of PLK1 led to an 85 percent reduction in breast tumors in mice that would normally have developed breast cancer due to a strong oncogene Kras or Her2. Combining excellent university hospitals with high-profile research at a Helmholtz Center is an important contribution to improving the chances of cancer patients. Cancer drug - in a different way than expected No. They discovered that the normal distribution of chromosomes into the two daughter cells is disturbed in the PLK1 overexpressing cells. Plk1 overexpression induces chromosomal instability and suppresses tumor development. Nature CommunicationsDOI: They develop novel approaches to make tumor diagnosis more precise and treatment of cancer patients more successful.

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    The results suggest that PLK1 is more of a tumor suppressor than a cancer-promoting oncogene, as previously suspected.

    The enzyme is therefore regarded as a promising target structure for new cancer drugs. Current Press releases Press releases archive. However, there are also contradictory results on the role of PLK1 in carcinogenesis.

    They discovered that the normal distribution of chromosomes into the two daughter cells is disturbed in the PLK1 overexpressing cells.

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    Other studies have shown that high PLK1 expression is even associated with a better prognosis in certain forms of breast cancer. The few tumors that developed despite PLK1 overexpression were significantly delayed.

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    Nature CommunicationsDOI: Combining excellent university hospitals with high-profile research at a Helmholtz Center is an important contribution to improving the chances of cancer patients. Using these animals, the researchers investigated the effects of high PLK1 levels on cell division and tumor development.

    At DKFZ, more than 1, scientists investigate how cancer develops, identify cancer risk factors and endeavor to find new strategies to prevent people from getting cancer. It regulates the formation of the division spindle and the correct distribution of chromosomes among the daughter cells. They discovered that the normal distribution of chromosomes into the two daughter cells is disturbed in the PLK1 overexpressing cells.

    The kinase PLK1 is overexpressed in a broad spectrum of human tumors.

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    Using these animals, the researchers investigated the effects of high PLK1 levels on cell division and tumor development.

    Many important components of the cell division apparatus are suitable as targets for innovative treatment approaches because the tumor cells are extremely dependent on these structures," explains the tumor biologist and adds: The results suggest that PLK1 is more of a tumor suppressor than a cancer-promoting oncogene, as previously suspected. However, there are also contradictory results on the role of PLK1 in carcinogenesis.

    It regulates the formation of the division spindle and the correct distribution of chromosomes among the daughter cells. Overexpression of PLK1 led to an 85 percent reduction in breast tumors in mice that would normally have developed breast cancer due to a strong oncogene Kras or Her2. However, scientists at the German Cancer Research Center have now discovered that high PLK1 levels in mice reduce the development of cancer and disrupt cell division.

    Cancer drug in a different way than expected

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    They discovered that the normal distribution of chromosomes into the two daughter cells is disturbed in the PLK1 overexpressing cells.

    At DKFZ, more than 1, scientists investigate how cancer develops, identify cancer risk factors and endeavor to find new strategies to prevent people from getting cancer. The kinase PLK1 is overexpressed in a broad spectrum of human tumors. The few tumors that developed despite PLK1 overexpression were significantly delayed. Several inhibitors that block the activity of PLK1 are currently undergoing clinical trials.

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    The enzyme is therefore regarded as a promising target structure for new cancer drugs.

    Combining excellent university hospitals with high-profile research at a Helmholtz Center is an important contribution to improving the chances of cancer patients.

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    They discovered that the normal distribution of chromosomes into the two daughter cells is disturbed in the PLK1 overexpressing cells. High PLK1 levels are associated with an unfavorable prognosis in many cancers, which is why PLK1 is considered a classic oncogene. Other studies have shown that high PLK1 expression is even associated with a better prognosis in certain forms of breast cancer. Using these animals, the researchers investigated the effects of high PLK1 levels on cell division and tumor development.

    However, PLK1 inhibitors can still work against tumors - only in a different way than expected.

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    Subscribe to our RSS-Feed. Overexpression of PLK1 led to an 85 percent reduction in breast tumors in mice that would normally have developed breast cancer due to a strong oncogene Kras or Her2.

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    Combining excellent university hospitals with high-profile research at a Helmholtz Center is an important contribution to improving the chances of cancer patients. The enzyme is therefore regarded as a promising target structure for new cancer drugs. The enzyme is therefore considered an oncogene and a promising target structure for new cancer therapeutics. A blockage of PLK1 results in incorrectly distributed chromosomes, stop of the cell cycle, and finally in cell death.

    The kinase PLK1 is overexpressed in a broad spectrum of human tumors.